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Anti-HCV Core protein/FITC,標(biāo)記的丙肝病毒核心抗原C區(qū)抗體

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研盟生物標(biāo)記抗體“Anti-HCV Core protein/FITC,標(biāo)記的丙肝病毒核心抗原C區(qū)抗體“，規(guī)格100ul，保證品質(zhì)，咨詢：

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產(chǎn)品名稱 Anti-HCV Core protein/FITC,標(biāo)記的丙肝病毒核心抗原C區(qū)抗體

產(chǎn)品編號(hào) BYK-0221R-FITC

英文名稱 Anti-HCV Core protein/FITC

中文名稱 FITC標(biāo)記的丙肝病毒核心抗原C區(qū)抗體

別名 Hepatitis C Virus antigen; Hepatitis C Virus Core 1a; Core protein p19; Hepatitis C Virus Core Antigen-C; HCVAg; HCV-Core protein;HCV core antigen; HCV core protein; Hepatitis C Virus core protein; capsid protein; polyprotein precursor; Core protein p21; Core protein p9.

研究領(lǐng)域細(xì)胞生物  免疫學(xué)  細(xì)菌及病毒
抗體來源 Rabbit
克隆類型 Polyclonal
交叉反應(yīng) HCV
產(chǎn)品應(yīng)用 IF=1:50-200
not yet tested in other applications.
optimal dilutions/concentrations should be determined by the end user.
分子量 19/21kDa
細(xì)胞定位細(xì)胞膜
性    狀 Lyophilized or Liquid
濃    度 1mg/ml
免疫原 KLH conjugated synthetic peptide derived from human HCV-Core 1-50aa

亞型 IgG
純化方法 affinity purified by Protein A
儲(chǔ) 存液 0.01M TBS(pH7.4) with 1% BSA, 0.03% Proclin300 and 50% Glycerol.
保存條件 Store at -20 °C for one year. Avoid repeated freeze/thaw cycles. The lyophilized antibody is stable at room temperature for at least one month and for greater than a year when kept at -20°C. When reconstituted in sterile pH 7.4 0.01M PBS or diluent of antibody the antibody is stable for at least two weeks at 2-4 °C.

Anti-HCV Core protein/FITC,標(biāo)記的丙肝病毒核心抗原C區(qū)抗體產(chǎn)品介紹：Core protein packages viral RNA to form a viral nucleocapsid, and promotes virion budding. Modulates viral translation initiation by interacting with HCV IRES and 40S ribosomal subunit. Also regulates many host cellular functions such as signaling pathways and apoptosis. Prevents the establishment of cellular antiviral state by blocking the interferon-alpha/beta (IFN-alpha/beta) and IFN-gamma signaling pathways and by inducing human STAT1 degradation. Plays an important role in virus-mediated cell transformation leading to hepatocellular carcinomas. Interacts with, and activates STAT3 leading to cellular transformation. May repress the promoter of p53, and sequester CREB3 and SP110 isoform3/Sp110b in the cytoplasm. Also represses cell cycle negative regulating factor CDKN1A, thereby interrupting an important check point of normal cell cycle regulation. Targets transcription factors involved in the regulation of inflammatory responses and in the immune response: suppresses NK-kappaB activation, and activates AP-1. Mediates apoptotic pathways throught association with TNF-type receptors TNFRSF1A and LTBR, although its effect on death receptors-induced apoptosis remains controvertial. Enhances TRAIL mediated apoptosis, suggesting that it might play a role in mediated apoptosis, suggesting that it might play a role in immune-mediated liver cell injury. Secreted core protein is able to bind C1QR1 at the T-cell surface, resulting in down-regulation of T-lymphocytes proliferation. May transactivate human MYC, Rous sarcoma virus LTR, and SV40 promoters. May suppress the human FOS and HIV-1 LTR activity. May alter lipid metabolism by interacting with hepatocellular proteins involved in lipid accumulation and storage.

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